[HSF] Aprotinin

Ben Bidstrup benjamin.bidstrup at bigpond.com
Mon Nov 20 11:16:58 EST 2006


You might find it interesting to read the FDA transcript of the 
Cardiovascular and Renal Advisory  Board 21 September.

>Please don't disparage Dennis Mangano too much.  He is, or at least was, a
>very capable clinical cardiac anesthesiologist and is fully cognizant of all
>of the issues regarding intra-operative bleeding and post-operative care of
>cardiac surgery patients.  That explains Dennis' consistent ability to focus
>and publish provocatively on real life issues that confronting surgeons and
>anesthesiologists on a day to day basis.  The methodology of his paper in
>the NEJM is open to question.  Dr. Mangano's credentials are not!
>Fraser Keith
>
>-----Original Message-----
>From: openheart-l-bounces at lists.hsforum.com
>[mailto:openheart-l-bounces at lists.hsforum.com] On Behalf Of Michael
>Firstenberg
>Sent: Sunday, November 19, 2006 10:18 AM
>To: OpenHeart-L at lists.hsforum.com
>Subject: Re: [HSF] Aprotinin
>
>If I recall Mangino is not a surgeon - in fact is he not an 
>anesthesiologist, as are many of the people who recently write these 
>articles about "bad cardiac drugs"?  Has he actually had to stand at 
>the foot of a bed or in the OR for countless hours watching patient 
>bleed to death and deal first hand with the consequences of massive 
>transfusions.  Yes, renal failure and dialysis is bad bad bad - but 
>compare that with right heart failure/ARDS/massive pressor 
>requirements/etc from excessive bleeding (and the hypotension and 
>associated ATN/renal failure anyhow).  My guess is he is home in bed 
>all nice an cozy with his pager off at the end of his shift.
>
>-michael
>
>
>
>
>On Nov 19, 2006, at 2:24 AM, Ani Anyanwu wrote:
>
>>  Prasanna
>>
>>  Well many would I suspect call it unbridled.
>>
>>  The following would generally receive aprotinin in my institution
>>  1) reoperations
>>  2) operations on the aortic arch or descending aorta
>>  3) transplant and VAD procedures
>>  4) operations on patients on clopidogrel
>>  5) combined valvular and CABG
>>  6) Patients with renal impairment
>>  7) Patients where ability to tolerate transfusion or bleeding 
>>  complications is thought to be marginal including - most patients 
>>  aged 70 or above, patients with severe lung disease, poor LV 
>>  function, severe pulmonary hypertension, multiple comorbidity etc. 
>>  Certainly almost all octogenrians would get aprotinin - even for CABG.
>>  8) Paradoxically, young patients in their 20s or 30s (where 
>>  avoidance of blood transfusion should be the goal in all patients)
>>  9) Multiple valvular procedures (excluding tricuspid valve)
>>  10) cases with anticipated bypass run more than 3 hours (including 
>>  complex mitral repairs)
>>
>>  As you can see there is not much left - so maybe it is unbridled! 
>>  As you implied we obviously would not use it for an ASD or isolated 
>>  AVR, but these constitute a small minority of our procedures. 
>>  Personally I would use it for practically every operation - 
>>  including all CABGs - but that is a personal opinion as I believe 
>>  there are non-hematological benefits of the drug and like you
>  > strongly believe in blood conservation. I do not have any interests 
>>  or links to industry.
>>
>>  Actually Ben brought up something that I had never thought of - 
>>  correct me if I am wrong but Aprotinin is the only agent licensed 
>>  as a blood conservation agent for heart surgery?
>>
>>  Ani
>>    ----- Original Message -----
>>    From: psimha<mailto:prasannasimha at gmail.com>
>>    To: OpenHeart-L at lists.hsforum.com<mailto:OpenHeart-
>>  L at lists.hsforum.com>
>>    Sent: Sunday, November 19, 2006 12:00 AM
>>    Subject: Re: [HSF] Aprotinin
>>
>>
>>    Ani - are you really using it "unbridled" or liberally ? Do you 
>>  use it
>>    for an ASD or for a straight forward valve replacement ? or any 
>>  other
>>    case with a short bypass run ?
>>    I did not say I will not use it in a redo - in fact if you note my
>>    original post I said I did use it in redo's ?
>>    And Yes , I believe very strongly in blood conservation and 
>>  believe that
>>    Aprotinin is one (and not the only ) cog in the wheel.
>  >   Prasanna
>>
>>    Ani Anyanwu wrote:
>>>  Prasanna
>>>
>>>  We use aprotinin in an unbridled way and are certainly yet to see 
>>>  this price.
>>>  - we have no more an incidence of renal failure than other 
>>>  institutions have (this we know because incidence of dialysis 
>>>  postop in all New York Hospitals is tracked by the State 
>>>  Department of Health)
>>>  - we have no suggestion of an increase in early vein graft 
>>>  thrombosis (this should transform into higher periop MI and 
>>>  mortality, our CABG mortality rate has remained around 1.5% last 3 
>>>  years)
>>>  - we have not experienced any adverse events that caused us to be 
>>>  concerned about its use, except fatal thrombosis in 2 patients 
>>>  with Factor V Lieden deficiency having circulatory arrest so we 
>>>  now routinely screen for this defect in all circulatory arrest cases.
>>>
>>>  The price we are paying is a low incidence of transfusion of blood 
>>>  products and a low re-exploration rate (<2% last 2 years even with 
>>>  18% being redos and almost 20% aortic cases). Maybe there are 
>>>  other unknown adverse effects which will catch up with us, but for 
>>>  know they are unknown (and we wont be responsible; remember it is 
>>>  the drug companies not doctors being sued for COX2 inhibitors).
>>>
>>>  Maybe when Mangano is bored he might do another study, and then 
>>>  what will you do? For those who use Amicar, how do we really know 
>>>  it is any safer - the drug is not even licensed for human use in 
>>>  many European countries. Perhaps even his next study will be on 
>>>  morbidity of plasma and platelet transfusions....then what will we 
>>>  do?
>>>
>>>  Ani
>>>    ----- Original Message -----
>>>    From: 
>>>  prasannasimha<mailto:prasannasimha at gmail.com<mailto:prasannasimha at gma
>>>  il.com>>
>>>    To: OpenHeart-L at lists.hsforum.com<mailto:OpenHeart-
>>>  L at lists.hsforum.com<mailto:OpenHeart-
>>>  L at lists.hsforum.com<mailto:OpenHeart-L at lists.hsforum.com>>
>>>    Sent: Saturday, November 18, 2006 9:37 PM
>>>    Subject: Re: [HSF] Aprotinin
>>>
>>>
>>>    The thing I want to say is that be it Vioxx / Aprotinin/blood/
>>>  Oxygen -
>>>    they are all drugs and have effects and side effects. The 
>>>  present mess
>>>    that the pharmacological companies are in is just because of their
>>>    unbridled enthusiasm (or greed) to ,make a quick buck and it 
>>>  backfires
>>>    on them. COX2 Inhibitors have a specific role unfortunately I 
>>>  even saw
>>>    my dentist prescribing it for tooth pain !! Who marketed it to 
>>>  him as a
>>>    good NSAID  ? I told him about the literature and my concerns 
>>>  (this was
>>>    prior to Vioxx) . They were trying to market Valdecoxib for post 
>>>  cardiac
>>>    surgery pain !!_ and I told them you should not be doing that - 
>>>  but did
>>>    they listen ? and bang in a few months a controversy breaks out. 
>>>  The
>>>    wife of colleague of mine was taking valdecoxib sample (she is a 
>>>  Doctor
>>>    too) as the sample was around and the premenopausal lady ended 
>>>  up with a
>>>    coronary thrombosis !!
>>>    Every drug has a role and an indication based on good clinical 
>>>  judgment
>  >>   - unfortunately we pay the price when its use is unbridled.
>>>    Prasanna
>>>   
>>>  hgrmd at aol.com<mailto:hgrmd at aol.com<mailto:hgrmd at aol.com<mailto:hgrmd@
>>>  aol.com>> wrote:
>>>>  Prasanna and Ajit,
>>>>    At the risk of great bodily harm from Ben, Ani, and others, I 
>>>>  again think the use of aprotinin should be limited as much as 
>>>>  possible.  I know there are cases where the benefit seemingly 
>>>>  outweighs the risk.  However, the mounting literature against it 
>>>>  is becoming increasingly compelling.  In addition, my own 
>>>>  impression, made years before any of this came out, was that the 
>>>>  drug increased the risk of ATN.  I'm also convinced that this has 
>>>>  the potential to be the Vioxx of cardiac surgery.  All I can say 
>>>>  is you guys who continue to indiscriminantly use it have got some 
>>>>  really big ones.
>>>>  Hal
>>>>
>>>>
>>>>  -----Original Message-----
>>>>  From: 
>>>>  prasannasimha at gmail.com<mailto:prasannasimha at gmail.com<mailto:prasan
>>>>  nasimha at gmail.com<mailto:prasannasimha at gmail.com>>
>>>>  To: OpenHeart-L at lists.hsforum.com<mailto:OpenHeart-
>  >>> L at lists.hsforum.com<mailto:OpenHeart-
>>>>  L at lists.hsforum.com<mailto:OpenHeart-L at lists.hsforum.com>>
>>>>  Sent: Sat, 18 Nov 2006 1:00 PM
>>>>  Subject: Re: [HSF] Aprotinin
>>>>
>>>>
>>>>  Very Sorry used Aprotinin on my redo - can't help using it 
>>>>  selectively !!
>>>>  Prasanna
>>>>
>>>>  Ajit Damle wrote:
>>>>
>>>>>  Journal club critique >
>>>>>  A disheartening story: Aprotinin in cardiac surgery >
>>>>>  Lien M, Milbrandt E
>>>>>
>>>>>  Critical Care, 2006 10:317 ( 8 November 2006 )
>>>>>
>>>>>
>>>>>  Journal club critique
>>>>>
>>>>>
>>>>>  A disheartening story: Aprotinin in cardiac surgery
>>>>>
>>>>>  Marcus Lien1 and Eric B Milbrandt2 >
>>>>>  1Clinical Fellow, Department of Critical Care Medicine, 
>>>>>  University of
>>>>>  Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USA
>>>>>
>>>>>  2Assistant Professor, Department of Critical Care Medicine, 
>>>>>  University of
>>>>>  Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, USA
>>>>>
>>>>>
>>>>>  Critical Care 2006, 10:317 doi:10.1186/cc5072
>>>>>
>>>>>
>>>>>>
>>>>>>
>>>>>  Evidence based medicine journal club critique edited by E B 
>>>>>  Milbrant
>>>>>
>>>>>
>>>>>  The electronic version of this article is the complete one and 
>>>>>  can be found
>>>>>  online at: http://ccforum.com/content/10/6/317<http://
>>>>>  ccforum.com/content/10/6/317<http://ccforum.com/content/
>>>>>  10/6/317<http://ccforum.com/content/10/6/317>>
>>>>>
>>>>>
>>>>>  Published 8 November 2006 >
>>>>>
>>>>>  C 2006 BioMed Central Ltd
>>>>>
>>>>>  Citation
>>>>>
>>>>>  Mangano DT, Tudor IC, Dietzel C: The risk associated with 
>>>>>  aprotinin in
>>>>>  cardiac surgery. N Engl J Med 2006, 354:353-365 [1].
>>>>>
>>>>>
>>>>>  Background
>>>>>
>>>>>
>>>>>  The majority of patients undergoing surgical treatment for ST-
>>>>>  elevation
>>>>>  myocardial infarction receive antifibrinolytic therapy to limit 
>>>>>  blood loss.
>>>>>  This approach appears counterintuitive to the accepted medical 
>>>>>  treatment of
>>>>>  the same condition - namely, fibrinolysis to limit thrombosis. 
>>>>>  Despite this
>>>>>  concern, no independent, large-scale safety assessment has been 
>>>>>  undertaken.
>>>>>
>>>>>
>>>>>  Methods
>>>>>
>>>>>
>>>>>  Design and setting
>>>>>
>>>>>
>>>>>  Prospective observational cohort study in 69 institutions in 
>>>>>  North and South
>>>>>  America, the Middle East, Europe, and Asia.
>>>>>
>>>>>
>>>>>  Subjects
>>>>>
>>>>>
>>>>>  4374 patients undergoing coronary-artery revascularization. All 
>>>>>  patients
>>>>>  were >18 years old and completed a pre-surgery interview. 
>>>>>  Patients were
>>>>>  classified as undergoing primary surgery (no previous heart 
>>>>>  surgery and no
>>>>>  other surgery besides a coronary artery bypass graft), or 
>>>>>  complex surgery
>>>>>  (all other surgery).
>>>>>
>>>>>
>>>>>  Intervention
>>>>>
>>>>>
>>>>>  None.
>>>>>
>>>>>
>>>>>  Measurements
>>>>>
>>>>>
>>>>>  The authors prospectively assessed three agents (aprotinin [1295 
>>>>>  patients],
>>>>>  aminocaproic acid [883], and tranexamic acid [822]) as compared 
>>>>>  with no
>>>>>  agent (1374 patients) with regard to serious cardiovascular,
>  >>>> renal, and
>>>>>  cerebrovascular outcomes by propensity and multivariable methods.
>>>>>
>>>>>
>>>>>  Results
>>>>>
>>>>>
>>>>>  In propensity-adjusted, multivariable logistic regression (C-
>>>>>  index, 0.72),
>>>>>  use of aprotinin was associated with a doubling in the risk of 
>>>>>  renal failure
>>>>>  requiring dialysis among patients undergoing complex coronary-
>>>>>  artery surgery
>>>>>  (odds ratio, 2.59; 95 percent confidence interval, 1.36 to 4.95) 
>>>>>  or primary
>>>>>  surgery (odds ratio, 2.34; 95 percent confidence interval, 1.27 
>>>>>  to 4.31).
>>>>>  Similarly, use of aprotinin in the latter group was associated 
>>>>>  with a 55
>>>>>  percent increase in the risk of myocardial infarction or heart 
>>>>>  failure (P <
>>>>>  0.001) and a 181 percent increase in the risk of stroke or 
>>>>>  encephalopathy (P
>>>>>  = 0.001). Neither aminocaproic acid nor tranexamic acid was 
>>>>>  associated with
>>>>>  an increased risk of renal, cardiac, or cerebral events. Adjustment
>>>>>  according to propensity score for the use of any one of the 
>>>>>  three agents as
>>>>>  compared with no agent yielded nearly identical findings. All 
>  >>>> the agents
>>>>>  reduced blood loss.
>>>>>
>>>>>
>>>>>  Conclusion
>>>>>
>>>>>
>>>>>  The association between aprotinin and serious end-organ damage 
>>>>>  indicates
>>>>>  that continued use is not prudent. In contrast, the less 
>>>>>  expensive generic
>>>>>  medications aminocaproic acid and tranexamic acid are safe 
>>>>>  alternatives.
>>>>>
>>>>>
>>>>>>
>>>>>  The medical and surgical approaches to acute ST-elevation 
>>>>>  myocardial
>>>>>  infarction present an interesting paradox. The medical approach 
>>>>>  focuses on
>>>>>  fibrinolytic therapy. Due to concerns over bleeding, the 
>>>>>  surgical approach
>>>>>  avoids fibrinolytic agents and instead uses agents that mitigate 
>>>>>  bleeding,
>>>>>  so called antifibrinolytic agents, which include aprotinin, 
>>>>>  aminocaproic
>>>>>  acid, and tranexamic acid. These agents were generally 
>>>>>  considered safe based
>>>>>  on a number of secondary analyses of studies that were not 
>>>>>  primarily
>>>>>  intended to assess safety. These relatively small studies were 
>>>>>  underpowered
>>>>>  to detect adverse events and did not involve head-to-head 
>>>>>  comparisons of the
>>>>>  commonly used antifibrinolytic agents. Animal studies suggest 
>>>>>  that these
>>>>>  agents have the potential to cause ischemic damage to multiple 
>>>>>  organ systems
>>>>>  and small, largely single-center studies have suggested 
>>>>>  increased graft
>>>>>  thrombosis and renal dysfunction [2-6]. Ideally, the safety of 
>>>>>  these agents
>>>>>  would be compared in a large, multi-center, randomized 
>>>>>  controlled trial.
>>>>>  However, because their use is embedded in practice and because 
>>>>>  regulatory
>>>>>  approval of these agents differs by country, conducting such a 
>>>>>  trial will be
>>>>>  difficult if not impossible.
>>>>>
>>>>>
>>>>>  To address the safety of these agents for cardiopulmonary bypass 
>>>>>  surgery,
>>>>>  Mangano and colleagues [1] conducted a large, prospective, 
>>>>>  observational
>>>>>  cohort assessing aprotinin, aminocaproic acid, and tranexamic 
>>>>>  acid as
>>>>>  compared to no agent in 4374 patients undergoing 
>>>>>  revascularization. Because
>>>>>  this was a prospective study, the authors were able to collect a 
>>>>>  wealth of
>>>>>  clinical information, including approximately 7500 data fields 
>>>>>  per patient.
>>>>>  This permitted consideration of variables that might influence 
>>>>>  both choice
>>>>>  of antifibrinolytic agent and clinical outcome. The authors used a
>>>>>  propensity score based on 45 treatment-selection covariates and
>>>>>  multivariable modeling to control for baseline differences 
>>>>>  between groups.
>>>>>  In doing so, they found that aprotinin, but not aminocaproic 
>>>>>  acid or
>>>>>  tranexamic acid, was associated with serious cardiovascular, 
>>>>>  renal, and
>>>>>  cerebrovascular adverse events. Furthermore, a dose-response 
>>>>>  relationship
>>>>>  was demonstrated, strengthening the inference of causality.
>>>>>
>>>>>
>>>>>  The main weakness of this study is that the authors failed to
>  >>>> report details
>>>>>  of the surgery itself, such as whether the surgery was on vs. 
>>>>>  off-pump, time
>>>>>  on pump, and number of vessels bypassed. These variables are 
>>>>>  likely to
>>>>>  influence not only choice of antifibrinolytic agent but also 
>>>>>  outcome, and
>>>>>  are, therefore, a source of indication bias that could reflect 
>>>>>  unfavorably
>>>>>  on aprotinin.
>>>>>
>>>>>
>>>>>  Based on the results of this study and those of another 
>>>>>  observational study
>>>>>  suggesting renal toxicity [7], the United States Food and Drug
>>>>>  Administration (FDA) held an advisory committee meeting 
>>>>>  September 21, 2006
>>>>>  to consider the cardiovascular safety of aprotinin. Because of 
>>>>>  concerns
>>>>>  about the methodology of the study by Mangano and colleagues and 
>>>>>  because it
>>>>>  was the only study to suggest cardiovascular adverse events [8], 
>>>>>  the
>>>>>  advisory committee concluded that there was insufficient 
>>>>>  evidence to support
>>>>>  changing the cardiovascular safety labeling of the drug. 
>>>>>  However, just six
>>>>>  days after the committee met, it was revealed that the drug's 
>>>>>  manufacturer,
>  >>>> Bayer, had preliminary results from an observational study of 
>>>>>  67,000 cardiac
>>>>>  bypass patients that suggested aprotinin was associated with 
>>>>>  increased risk
>>>>>  of death, renal dysfunction, congestive heart failure, and 
>>>>>  stroke [9]. The
>>>>>  FDA subsequently issued a statement indicating it was unaware of 
>>>>>  this study
>>>>>  when the advisory committee met and that it is evaluating the 
>>>>>  results of
>>>>>  this study and the potential implications for the use of 
>>>>>  aprotinin [10]. In
>>>>>  the mean time, the FDA suggests that physicians who use 
>>>>>  aprotinin should
>>>>>  carefully monitor patients for the occurrence of toxicity, 
>>>>>  particularly to
>>>>>  the kidneys, heart, or brain, and promptly report observed 
>>>>>  adverse events.
>>>>>  They go on to recommend that physicians should consider limiting 
>>>>>  aprotinin
>>>>>  use to those situations where the clinical benefit of reduced 
>>>>>  blood loss is
>>>>>  essential to medical management of the patient and outweighs the 
>>>>>  potential
>>>>>  risks.
>>>>>
>>>>>
>>>>>  Recommendation >
>>>>>
>>>>>  The weight of evidence suggests that aprotinin increases the 
>>>>>  risk for a poor
>>>>>  outcome among patients undergoing cardiac operations. Not only 
>>>>>  is this drug
>>>>>  very expensive, it seems to be toxic. Although the risk of 
>>>>>  excessive
>>>>>  bleeding is certainly a cause for concern in certain patients, 
>>>>>  and treatment
>>>>>  with aprotinin can decrease blood loss in selected patients, 
>>>>>  data are
>>>>>  lacking to show that administration of this agent actually improves
>>>>>  survival.
>>>>>
>>>>>
>>>>>  Competing interests
>>>>>
>>>>>  The authors declare that they have no competing interests.
>>>>>
>>>>>
>>>>>>
>>>>>  1. Mangano DT, Tudor IC, Dietzel C: The risk associated with 
>>>>>  aprotinin in
>>>>>  cardiac surgery.
>>>>>
>>>>>  N Engl J Med 2006, 354:353-365. >
>>>>>
>>>>>  2. Cosgrove DM III, Heric B, Lytle BW, Taylor PC, Novoa R, 
>>>>>  Golding LA,
>>>>>  Stewart RW, McCarthy PM, Loop FD: Aprotinin therapy for reoperative
>>>>>  myocardial revascularization: a placebo-controlled study.
>>>>>
>>>>>  Ann Thorac Surg 1992, 54:1031-1036.
>>>>>
>>>>>
>>>>>  3. D'Ambra MN, Akins CW, Blackstone EH, Bonney SL, Cohn LH, 
>>>>>  Cosgrove DM,
>>>>>  Levy JH, Lynch KE, Maddi R: Aprotinin in primary valve 
>>>>>  replacement and
>>>>>  reconstruction: a multicenter, double-blind, placebo-controlled 
>>>>>  trial.
>>>>>
>>>>>  J Thorac Cardiovasc Surg 1996, 112:1081-1089
>>>>>
>>>>>
>>>>>  4. Feindt PR, Walcher S, Volkmer I, Keller HE, Straub U, Huwer 
>>>>>  H, Seyfert
>>>>>  UT, Petzold T, Gams E: Effects of high-dose aprotinin on renal 
>>>>>  function in
>>>>>  aortocoronary bypass grafting.
>>>>>
>>>>>  Ann Thorac Surg 1995, 60:1076-1080 >
>>>>>
>>>>>  5. Sundt TM III, Kouchoukos NT, Saffitz JE, Murphy SF, Wareing 
>>>>>  TH, Stahl
>>>>>  DJ: Renal dysfunction and intravascular coagulation with 
>>>>>  aprotinin and
>>>>>  hypothermic circulatory arrest.
>  >>>>
>>>>>  Ann Thorac Surg 1993, 55:1418-1424 >
>>>>>
>>>>>  6. Umbrain V, Christiaens F, Camu F: Intraoperative coronary 
>>>>>  thrombosis:
>>>>>  can aprotinin and protamine be incriminated?
>>>>>
>>>>>  J Cardiothorac Vasc Anesth 1994, 8:198-201 >
>>>>>
>>>>>  7. Karkouti K, Beattie WS, Dattilo KM, McCluskey SA, Ghannam M, 
>>>>>  Hamdy A,
>>>>>  Wijeysundera DN, Fedorko L, Yau TM: A propensity score case-control
>>>>>  comparison of aprotinin and tranexamic acid in high-transfusion-
>>>>>  risk cardiac
>>>>>  surgery.
>>>>>
>>>>>  Transfusion 2006, 46:327-338 >
>>>>>
>>>>>  8. Hughes S: Aprotinin safety again in spotlight as new study 
>>>>>  suggests
>>>>>  increased cardiac events.
>>>>>
>>>>>  http://www.medscape.com/viewarticle/545400<http://
>>>>>  www.medscape.com/viewarticle/545400<http://www.medscape.com/
>>>>>  viewarticle/545400<http://www.medscape.com/viewarticle/545400>> >
>>>>>  October 2, 2006 >
>>>>>  9. Harris G: FDA says Bayer failed to reveal drug risk study.
>>>>>
>>>>>  [http://www.nytimes.com/2006/09/30/health/30fda.html] New York 
>>>>>  Times >
>>>>>
>>>>>  10. US Food and Drug Administration: FDA Public Health Advisory: 
>>>>>  Aprotinin
>>>>>  Injection (marketed as Trasylol).
>>>>>
>>>>>  [http://www.fda.gov/cder/drug/advisory/aprotinin20060929.htm] >
>  >>>> September 29, 2006 >
>>>>>
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>>>    http://www.hsforum.com/listdisclaim<http://www.hsforum.com/
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>>>  http://mmp.cjp.com/mailman/listinfo/openheart-l<http://mmp.cjp.com/
>  >> mailman/listinfo/openheart-l>
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>>>  policies and
>>>  disclaimers posted at:
>>>  http://www.hsforum.com/listdisclaim<http://www.hsforum.com/
>>>  listdisclaim>
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>>
>>    _______________________________________________
>>    OpenHeart-L mailing list
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>>    Send postings to:
>>     OpenHeart-L at lists.hsforum.com<mailto:OpenHeart-L at lists.hsforum.com>
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>>    To UNSUBSCRIBE, to CHANGE email address, or to view archives:
>>    http://mmp.cjp.com/mailman/listinfo/openheart-l<http://
>>  mmp.cjp.com/mailman/listinfo/openheart-l>
>>
>>    All messages transmitted by the OpenHeart-L are subject to the 
>>  policies and
>>    disclaimers posted at:
>>    http://www.hsforum.com/listdisclaim<http://www.hsforum.com/
>>  listdisclaim>
>>    -----------------------------------------
>>  _______________________________________________
>>  OpenHeart-L mailing list
>>
>>  Send postings to:
>>   OpenHeart-L at lists.hsforum.com
>>
>>  To UNSUBSCRIBE, to CHANGE email address, or to view archives:
>>  http://mmp.cjp.com/mailman/listinfo/openheart-l
>>
>>  All messages transmitted by the OpenHeart-L are subject to the 
>>  policies and
>>  disclaimers posted at:
>>  http://www.hsforum.com/listdisclaim
>>  -----------------------------------------
>
>_______________________________________________
>OpenHeart-L mailing list
>
>Send postings to:
>  OpenHeart-L at lists.hsforum.com
>
>To UNSUBSCRIBE, to CHANGE email address, or to view archives:
>http://mmp.cjp.com/mailman/listinfo/openheart-l
>
>All messages transmitted by the OpenHeart-L are subject to the policies and
>disclaimers posted at:
>http://www.hsforum.com/listdisclaim
>-----------------------------------------
>
>_______________________________________________
>OpenHeart-L mailing list
>
>Send postings to:
>  OpenHeart-L at lists.hsforum.com
>
>To UNSUBSCRIBE, to CHANGE email address, or to view archives:
>http://mmp.cjp.com/mailman/listinfo/openheart-l
>
>All messages transmitted by the OpenHeart-L are subject to the policies and
>disclaimers posted at:
>http://www.hsforum.com/listdisclaim
>-----------------------------------------


-- 
Ben Bidstrup FRACS FRCSEd FEBCTS
Consultant Cardiothoracic Surgeon


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