AW: [HSF] Deairing the heart
Rwmfglycar at aol.com
Rwmfglycar at aol.com
Fri Mar 2 23:18:55 EST 2007
In a message dated 3/2/2007 8:13:27 A.M. Eastern Standard Time,
anianyanwu at hotmail.com writes:
But Hal I would question the relevance of subtle neurological changes that
require a battery of sophisticated tests to demonstrate. A lot of these
changes IMHO are integral to having artificial extracorporeal circulation and
indeed MRI studies show changes in many patients with no neurological
complications. Air and embolism are an integral part of CBP and in most cases are likely
of no or of minimal consequence. Obviously though we must exclude gross air
bubbles and our deairing approach, including CO2 and TEE is very similar to
yours.
.
However, I think to a great degree air is an invention of TEE and most of
what we see on TEE or strive to achieve with TEE is of little relevance above
that which can be determined clinically as in Novick's practice or in the
practice of the 1980s. Like you I vent the root up to 15 mins after coming of
bypass and I suspect in most patients this will be sufficient to deal with any
rogue bubbles that failed deairing - regardless of the presence of TEE.
I am not sure there is any direct evidence that the incidence of
neurological complication or neuropsychometric deficit has changed with the advent of
TEE or that the incidence is different in centers that routinely use TEE and
centers that don't. Those surgeons in life courses who don't seem to deair
properly may not necessarily be harming their patients. In my previous center
(Harefield with Yacoub) we generally ignored the snowstorms seen on TEE and
apparently (I know I know) to no consequence
Dear Ani,
When I had residents who expressed views like yours I would ask them if they
knew of any serious consequence of massive air embolism. They said yes. I
then asked how much air it took to produce the appearance of air seen on
echocardiography. They would not know but said it must be less than one would see
in a case of massive air embolism. I agreed but then asked if they thought
that 1 cc of air would be much less than massive air embolism.They were sure of
that. I would then ask them if they would let me inject 1cc of air up one of
their carotids. They would not. I then told them that until they had proven
conclusively that a certain quantity of air correlated with a certain density
of bubbles in the cardiac chambers did NOT produce any cerebral
consequences, it was unethical not to clear the air seen on echocardiography from the
cardiac chambers before coming of bypass.
Studies were done on dogs of the consequences of injecting 1 cc increments
of air into their carotids. Clinical consequences were evident with very small
amounts of air. I had an inverted U tube between the ascending aorta and the
caval cannulae for 20 years. The large bubbles that would collect while the
echo appearance of air dissipated were impressive.
You are in fact doing various maneuvers to get rid of air but at the same
time state "Air and embolism are an integral part of CBP and in most cases
are likely of no or of minimal consequence" . If you really believe that why
bother to deair? You also invoke your time with Magdi: "In my previous center
(Harefield with Yacoub) we generally ignored the snowstorms seen on TEE and
apparently (I know I know) to no consequence". If you are aware that this is
rubbish evidence don't present it. I have seen the snowstorms at Harefield and
I got the impression that you were too busy to have time to evaluate subtle
changes in your patients' cerebral function, in the postop period.
Cerebral dysfunction after extracorporeal circulation does occur and has
many possible causes. Air is one obvious cause that we can detect and do
something about. The evaluation of cerebral function remains a weak branch of
medicine.The fact that, because of that weakness, we cannot state that air removal
is not important but do know that it may be important obligates us to do
something about it.
Bob
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